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112,113 Moreover, given the multiplicity of 5-HT 2A receptor-expressing GABAergic interneurons on principal cells, eg, in the amygdala and hippocampus, 107 it seems likely that the effects of pharmacological treatments that either activate or inhibit 5-HT 2A receptors are overwhelmingly mediated by downstream effects of GABA neurotransmission
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This stimulation in the respective genes expression is possibly a part of the same mechanism to provide appropriate antioxidant defense by ensuring relevant GSH levels under conditions of oxidative stimulation by UA
Glutathione and various synergy vitamins work together to build an antioxidant network that delivers glutathione more efficiently for oxidation
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