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In only three cases did vB12 treatment rebalance a metabolic alteration observed in untreated Tbx1 +/− mice, namely, inosine, glutamate, and short-chain fatty acids (SCFAs) (Figs 4C and S3B)

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59,72,73 One study in pain-sensitized mice found neutrophils invaded peripheral sensory nerves and increased their pain signaling, whereas neutrophil depletion prevented the mice from developing chronic widespread pain