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Hou et al (79) found that AS-IV administration could promote the inactivation of NLRP3 and the fibrosis-related effector proteins collagen I, collagen II and α-SMA in TGF-β-induced PF, thereby alleviating epithelial-mesenchymal transition (EMT)

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This approach represents the first attempt to integrate immunotherapy with direct ECM degradation via collagenase, departing from conventional nanoparticle-based delivery of small molecules or antibody drugs to the tumor ECM