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The involvement of miR-25 in the regulation of apoptotic marker genes via downregulation followed by recovery of response upon reconstitution, as well as the EGCG-mediated induction of apoptosis and interference with cell cycle progression (G2/M block), provide evidence that miR-25 may be a regulator of the apoptotic effects created by EGCG in these cells

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Separate statistical analyses were used to: 1) compare baseline values between treatment groups, 2) compare post-treatment treatment differences in concentration for each glutathione parameter from respective baseline values across all time points, and 3) compare maximum post-treatment change from baseline for each glutathione parameter