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Another fusion protein targeting PD-L1 and TGF-β, SHR1701, is being investigated in a phase II trial in combination with cyclin-dependent kinase 4/6 inhibitor in patients with hormone receptor-positive, HER2-negative, endocrine-resistant advanced breast cancer (NCT04355858)
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SAMTOR directly senses SAM, and SAM depletion inhibits mTORC1 signaling, providing a parallel mechanism by which altered methyl-donor metabolism could limit growth
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