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C., “Molecular Mechanisms in Chaperonopathies: Clues to Understanding the Histopathological Abnormalities and Developing Novel Therapies,” Journal of Pathology 250, no
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In in vitro and in vivo experiments, anle138b blocked the formation of pathological aggregates of prion protein and of α-syn and inhibited oligomer accumulation, neuronal degeneration, and disease progression [134]
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