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Effects of an effective concentration of nicotine (N 250 μg/ml) in the presence or absence of glutathione (G 1–4 μg/ml) on the metabolism of 14C-testosterone and 14C-4-androstenedione by HPF (Figures 5 & 6) When 14C-testosterone was used as substrate (Figure 5), the main metabolites formed were Diols, DHT and androstanedione (A), of which DHT and A are shown

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This process involves various pathogenesis mechanisms, such as pathological calcium signaling in pancreatic acinar cells, changes in pH, coexistence and autophagy, the cleavage of trypsinogen by the lysosomal hydrolytic enzyme tissue protease B to trypsin, and decreased activity of the pancreatic acinar cell trypsin inhibitor (11-13)